Autism: An Introduction to Psychological Theory by Francesca Happé

By Francesca Happé

This name is meant for complex undergraduate and masters point scholars in psychology; expert medical, developmental and academic psychologists; common practitioners and others with a distinct curiosity in little ones; careworkers; and fogeys of autistic teenagers.

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The Sally-Ann task) does not reflect a lack of mentalizing but rather some other impairment, or task artifact; (b) those which accept that failure on false belief tasks reflects a lack of mentalizing, but deny that this is the primary, core psychological impairment on the basis of (i) lack of universality of this deficit, (ii) lack of causal precedence of this deficit. Does failure on false belief tasks reflect inability to mentalize? The false belief paradigm was originally proposed as a test safe from false positives (Dennett 1978).

10). It is possible, then, that the gene responsible for autism may in some cases produce far milder handicaps. Szatmari & Jones (1991) have recently discussed the types of inheritance that might be involved in autism. e. inherited from both father and mother); X linked (carried on the female sex chromosome). However, they stress that more data need to be collected concerning the pattern of occurrence of autism in families and the severity of attendant general learning difficulties, in order to explore the possible modes of inheritance.

1988). This could be easily explained by a model in which autism results from damage to a circumscribed brain region or pathway, call it X. Widespread damage to the brain, such as causes mental retardation, will be more likely to knock out the specific component X the more of the brain it affects. While no unambiguous and universal findings have emerged to suggest the site of the lesion, or the precise nature of the neurochemical pathway disrupted in autism, we can be confident that autism has a primary cause at the level of the brain (Steffenburg & Gillberg 1990).

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