By Takashi Tokoro
Pathologic myopia that's because of axial elongation motives thinning of the retina and choroid, in particular in circumstances of posterior staphyloma. the results of this myopia is the improvement of assorted sorts of chorioretinal atrophy within the posterior pole, with a gentle development of the atrophic alterations extending over numerous many years. beforehand, reviews of atrophic lesions and their medical direction were incomplete, and diagnostic criteria and type of chorioretinopathy were doubtful. The Atlas of PosteriorFundus adjustments in Pathologic Myopia discusses those and different vital questions about the foundation of long term commentary and learn. an immense function of the ebook is the presentation of many case reports, with beneficiant use of full-color photos to teach intimately the process fundus adjustments. The atlas is effective source not just for ophthalmologists drawn to myopia yet for optometrists, opticians, and clinical students.
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Extra resources for Atlas of Posterior Fundus Changes in Pathologic Myopia
Or Inferior Crescent Eyl's With Temporal. asal. or Inferior Crescent Eyes With Allilu lar ~ U Fig. 15. Shape of crescents in eyes with or without lacquer crack (Lc) lesions and no peripapillary atrophy. 4%, respectively (there is no significant difference) (Fig. 17). Therefore, it is understood that the peripapillary crescent will become annular if the chorioretinal atrophy is severe. 2 mm, respectively. Therefore, it is reasonable that the percentage of annular crescent will increase in eyes with aging and long axial length.
51). The percentage of Lc in eyes without staphyloma is as low as 5%-10% and does not show a distribution of two peaks. Conversely, the frequency distribution of Lc in eyes with staphyloma shows two peaks at around the age of 40 years and at about age 60 years. In other words, this finding reflects the characteristics of frequency distribution of Lc with age in eyes with posterior staphyloma. 1% (1 eye) had MA (Fig. 52). 0% per 10 years, and the rate of increase of eyes with MA was 0% per 10 years (Fig.
The incidence of tessellated fundus in eyes under age 30 years is quite rare, and it stabilizes to a low percentage in those over age 40. A dramatic reduction in the frequency of DI occurs linearly over age 30. The frequency of D2 is low under age 20 years, but increases sharply in those above age 25, and continues to increase, albeit at a slow pace (Fig. 49). >. JO 0 >. ::; -E. § 70 GO 50 '0 en tlO w ~ &h 8.. ~ g ~ 2 ~~ r: l- - = - I- i::l - - :::ll - • MA I""" .... " ,... ; [ I"7i ~. ,... :. - - r- - 0 0,,1 5·9 10· 14 15· 20 19 24 25 30· 35· 40· ,15, 50· 55 - 60· 29 34 39 '1'1 49 54 59 6'1 65 & above Age (Years) Fig.